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You prove my point. With all the research done on
this chemical ... why isn't it suspected for anything?
Yes it causes prostate cancer & related issues
... it causes many rashes (many are signs of autoimmune issues)
I suspect it is the most probable cause of
AUTISM and Non Hodgkin's Lymphoma, too
and you can skip 'metastasizing'
it is the 'shrapnel' chemical and can cause all of the damage,
all by itself. Where it shows up first is irrelevant
Note the article below. Yes, I suspect the
neurotoxin, organic solvent, pesticide, teratogen chemical for Autism
2-butoxyethanol or
BUTYL
same increases to quickly for NHL also
They say
NHL has increased by 50% ... way too dramatic to be a
hereditary issue alone - having an 'epidemic' of diabetes &
obesity also in USA, Britain, WWII European countries. Is it
often coupled with the hard to find anemia? AIHA or IMHA
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California's Autism Increase Not Due To Better
Counting, Diagnosis
Science Daily (Jan. 11, 2009) — A study by researchers
at the UC Davis M.I.N.D. Institute has found that the seven- to
eight-fold increase in the number children born in California with
autism since 1990 cannot be explained by either changes in how the
condition is diagnosed or counted — and the trend shows no sign of
abating.
Published in the January 2009 issue of the journal
Epidemiology, results from the study also suggest that
research should shift from
genetics to the host of chemicals and infectious microbes in the
environment that are likely at the root of changes in the
neurodevelopment of California's children.
"It's time to start looking for the environmental
culprits responsible for the remarkable increase in the rate of
autism in California," said UC Davis M.I.N.D. Institute researcher
Irva Hertz-Picciotto, a professor of environmental and occupational
health and epidemiology and an internationally respected autism
researcher.
Hertz-Picciotto said that many researchers, state
officials and advocacy organizations have viewed the rise in
autism's incidence in California with skepticism.
The incidence of autism by age six in California has
increased from fewer than nine in 10,000 for children born in 1990
to more than 44 in 10,000 for children born in 2000. Some have
argued that this change could have been due to migration into
California of families with autistic children, inclusion of children
with milder forms of autism in the counting and earlier ages of
diagnosis as consequences of improved surveillance or greater
awareness.
Hertz-Picciotto and her co-author, Lora Delwiche of
the UC Davis Department of Public Health Sciences, initiated the
study to address these beliefs, analyzing data collected by the
state of California Department of Developmental Services (DDS) from
1990 to 2006, as well as the United States Census Bureau and state
of California Department of Public Health Office of Vital Records,
which compiles and maintains birth statistics.
Hertz-Picciotto and Delwiche correlated the number of
cases of autism reported between 1990 and 2006 with birth records
and excluded children not born in California. They used Census
Bureau data to calculate the rate of incidence in the population
over time and examined the age at diagnosis of all children ages two
to 10 years old.
The methodology eliminated migration as a potential
cause of the increase in the number of autism cases. It also
revealed that no more than 56 percent of the estimated 600-to-700
percent increase, that is, less than one-tenth of the increased
number of reported autism cases, could be attributed to the
inclusion of milder cases of autism. Only 24 percent of the increase
could be attributed to earlier age at diagnosis.
"These are fairly small percentages compared to the
size of the increase that we've seen in the state," Hertz-Picciotto
said.
Hertz-Picciotto said that the study is a clarion call
to researchers and policy makers who have focused attention and
money on understanding the genetic components of autism. She said
that the rise in cases of autism in California cannot be attributed
to the state's increasingly diverse population because the disorder
affects ethnic groups at fairly similar rates.
"Right now, about 10 to 20 times more research
dollars are spent on studies of the genetic causes of autism than on
environmental ones. We need to even out the funding," Hertz-Picciotto
said.
The study results are also a harbinger of things to
come for public-health officials, who should prepare to offer
services to the increasing number of children diagnosed with autism
in the last decade who are now entering their late teen years,
Hertz-Picciotto said.
"These children are now moving toward adulthood, and
a sizeable percentage of them have not developed the life skills
that would allow them to live independently," she said.
The question for the state of California, Hertz-Picciotto
said, will become: 'What happens to them when their parents cannot
take care of them?'
"These questions are not going to go away and they
are only going to loom larger in the future. Until we know the
causes and can eliminate them, we as a society need to provide those
treatments and interventions that do seem to help these children
adapt. We as scientists need to improve available therapies and
create new ones," Hertz-Picciotto said.
Hertz-Picciotto and her colleagues at the M.I.N.D
Institute are currently conducting two large studies aimed at
discovering the causes of autism. Hertz-Picciotto is the principal
investigator on the CHARGE (Childhood Autism Risk from Genetics and
the Environment) and MARBLES (Markers of Autism Risk in
Babies-Learning Early Signs) studies.
CHARGE is the largest epidemiologic study of reliably
confirmed cases of autism to date, and the first major investigation
of environmental factors and gene-environment interactions in the
disorder. MARBLES is a prospective investigation that follows women
who already have had one child with autism, beginning early in or
even before a subsequent pregnancy, to search for early markers that
predict autism in the younger sibling.
"We're looking at the possible effects of metals,
pesticides and infectious agents on neurodevelopment," Hertz-Picciotto
said. "If we're going to stop the rise in autism in California, we
need to keep these studies going and expand them to the extent
possible."
The study was funded by grants from the National Institute of
Environmental Health Sciences (NIEHS) and by the M.I.N.D. Institute.
http://www.sciencedaily.com/releases/2009/01/090108095429.htm
Adapted from materials provided by University of California - Davis
- Health System.
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A study by researchers at the UC Davis M.I.N.D.
Institute has found that the seven- to eight-fold increase in the
number children born in California with autism since 1990 cannot be
explained by either changes in how the condition is diagnosed or
counted — and the trend shows no sign of abating.
Published in the January 2009 issue of the journal Epidemiology,
results from the study also suggest that
research should shift from
genetics to the host of chemicals and infectious microbes in the
environment that are likely at the root of changes in the
neurodevelopment of California's children.
"It's time to start looking for the environmental
culprits responsible for the remarkable increase in the rate of
autism in California,"
My thoughts:
I suspect the neurotoxin, organic solvent,
pesticide, teratogen chemical for Autism
2-butoxyethanol or
BUTYL
I suspect it is the cause of
AUTISM
and autoimmune issues in general
Text Version http://www.ucdmc.ucdavis.edu/medresearch/medsp/includes/SOMGrants2007.pdf |